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  • 1.
    Serotonin - Wikipedia, the free encyclopedia
  • Serotonin (pronounced /ˌsɛrəˈtoʊnən/) (5-hydroxytryptamine, or 5-HT) is a ... 2.3 Endothelial cell function and Serotonin. 3 Biosynthesis. 4 Drugs targeting ...
  • http://en.wikipedia.org/wiki/Serotonin
  • 2.
    serotonin: Definition from Answers.com
  • serotonin n. An organic compound, C 10 H 12 N 2 O, formed from tryptophan and found in animal and human tissue, especially the brain, blood serum,
  • http://www.answers.com/topic/serotonin
Questions/Answers
Serotonin?
Does anyone know a lot about MDMA? I'm doing some research on this topic, and have a couple questions: How long does it take for serotonin to replenish itself after having been completely drained?...And are there any good ways to hurry this process along? I know that a balanced diet with exercise is best...But, is there anything else? Does anyone know of any studies that have been done regarding long time usage of MDMA? And, can you ever predict what sort of other substances (speed, acid, caffeine, etc) might be in a pill? In other words, is it ever possible to get a certain kind, or is it all chance?
MDMA is a controlled substance, and class A drug in the UK. It is the active ingredient in Ecstasy. I know a little about the substance because it is used quite a lot on the club circuit, or at least was 2/3 years ago when I used to go out. Each person will have a different response to the release of serotonin in the brain, but I personally think that the constant use of a drug of this kind can actually cause clinical depression, where the body cannot normally function due to abuse. 'Speed' is also a controlled drug. If you were a recreational user of MDMA, I'd say the only way that the body could replenish, is a rest from using the drug naturally, without any other substance. The body's best chance of replenishing it's used up serotonin wouldn't be the use of another controlled drug, but a natural rest from using the substance in the first place. There is the 'coming down' where the next day, the person experiences (or can experience, it's different for each person) a depression-like state. I think that there are certain anti-depressants that a doctor would administer, that might be able to help, but you'd have to have those prescribed, and I wouldn't know where to start on this forum to begin to explain. A doctor in the UK wouldn't give any of these sort of drugs to a person that was abusing drugs in the first place. A friend of mine was a cocaine addict who informed his doctor of his addiction. He was going to Narcotics Anonymous and that meant that he had to be completely honest so as to not get any sort of drugs that might hinder his recovery from a doctor. Also, you can never know what is in a pill. There might even be heroin inside there. You have no idea what you are putting in your body. There is an old saying I know. "I'd rather eat my own s**t. It's cleaner" I can't say that I have known many people that have died from abusing recreational drugs, apart from a few overdoses that were intentional, but they really ruin your life if they take a hold of you, and I've seen some really lovely people turn into monsters from using these drugs.
Are there natural sources ofserotonin and/ornon-prescription ways tocreate or retain it the brain?
I'm not lookin for herbal remedies that elevate mood, just want to know if you can generate or maintain serotonin levels within your own body somehow (example excercise?) and/or if you can eat foods (not herbal supplements) that provide or create or maintain serotonin levels.
Exercise. meditation, cognitive therapy are my ways/
How long does it take for yourbody to stop making serotoninon antidepressant ?
I know if you take it too long, your body stops making its own serotonin, but how long is that? In psychology class, our teacher told us that your body slows the production of serotonin since the medicine is increasing it.
There is plenty of misleading information on this question. First, antidepressants affect brain serotonin (that's why they are prescribed). However, your teacher is misinterpreting basic neurophysiology and neuropharmacology. Clinically depressed patients have a decrease in the functional activity of monoamines, including serotonin (the other monoamines include norepinephrine and dopamine). It is pretty clear from the last 30+ years of clinical research that depression is directly related to serotonin in one of several ways: either not enough serotonin is being produced and/or released, or there is down regulation of serotonin receptor density or down regulation of serotonin receptor sensitivity. Common classes of antidepressants (e.g., SSRIs, MAOIs) remedy this by increasing the functional activity of the serotonin system. This is accomplished different ways but basically they each turn off the brain's natural turning off mechanism to make whatever serotonin is available more effective. Here's how: Normally, many neurochemicals when released from neurons make brief contact with nearby receptors which in turn either cause an increase or decrease in release of some other neurochemical stored in that neuron. This process is often turned off by something called reuptake (wherein the neurotransmitter is removed from the synapes so it cannot continue to interact with receptors). This ballet occurs millions of times a second in the brain. SSRIs block the reuptake (removal) of serotonin. If reuptake back into the neuron is blocked, it allows more serotonin-receptor interaction. Similarly, MAOIs accomplish the same end result but in a different way. Another way the brain turns off serotonin (and some other neurotransmitters) is through enzymatic destruction by an enzyme called monoamine oxidase) MAO). MAO inhibitors (MAOIs) inhibit MAO. By inhibiting the enzyme that breaks down serotonin, more serotonin is available to interact with serotonin receptors). None of these actions would decrease the manufacture of serotonin (and yes, I have considered presynaptic inhibitory feedback loops). It would make little sense if antidepressants decreased the ability of the brain to synthesize serotonin since the lack of serotonin (as discussed above) is an important cause of the depression being treated by the antidepressant! That thinking is simply illogical. Sorry.
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